Barry Robert, D.V.M., Ph.D.
Associate Professor - Research
Comparative Biology Core
Ph.D., Departments of Comparative Medicine and Pathology, Schools of Medicine and Dentistry, University of Alabama at Birmingham, Birmingham, Alabama, 1988, Pathology
D.V.M., Louisiana State University School of Veterinary Medicine, Baton Rouge, Louisiana, 1987
B.S., Louisiana State University, Baton Rouge, Louisiana, 1983, Microbiology
Dr. Robert’s research interest focuses on mechanisms leading to fibrosis and thickening of the peritoneal membrane and ultimately its failure to function as a dialysis membrane in kidney failure patients undergoing chronic peritoneal dialysis. The most common causes of kidney failure are diabetes mellitus, hypertension, and glomerulonephritis. Kidney failure patients are treated either with renal replacement therapy or a kidney transplant when a donor is available. Peritoneal dialysis serves as one form of renal replacement therapy. Over time many patients undergoing peritoneal dialysis experience failure of this mode of therapy due to fibrosis and thickening of the peritoneal membrane.
Abrahamson D.R., Robert B. Derivation and differentiation of glomerular endothelial cells. Nephrology Dialysis Transplantation. 18 Suppl 6:vi2-7, 2003.
Abrahamson, D.R., A.C. Prettyman, B. Robert, and P.L. St. John. Laminin-1 Reexpression in Alport mouse glomerular basement membranes. Kidney Int. 63(3): 826-834, 2003.
Freeburg, P.B., B. Robert, and D.R. Abrahamson. Podocyte expression of hypoxia-inducible factors (HIFs) –1 and –2 during glomerular development. J. Am. Soc. Nephrol. 14(4): 927-938, 2003.